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In tumor tissues, mTORC1 phosphorylation was reduced, and AMPK phosphorylation and LC3BII/I ratios were decreased in the TER group compared to other tumor-bearing groups, suggesting that the combined therapy not only helped preserve muscle mass but also altered tumor metabolic signaling pathways in a favorable direction (88)

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243 In AML, the level of H3K36me3 changes most dramatically before and after Met deprivation, and inhibition of STED2, the H3K36-specific methyltransferase, can reproduce most of the cytotoxic phenotypes produced by Met deprivation

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